Neutrophil extracellular traps (NETs) represent a distinct mechanism to control and eliminate
microbial infections. Our results show that conidia and germ tubes of the human pathogenic
mold Aspergillus fumigatus are able to trigger the formation of NETs. Viable fungal cells are
not essentially required for this host–pathogen interaction. Neutrophils engulf conidia and
thereby inhibit their germination, a process that is independent of NETosis. In the
experimental set-up used in this study neutrophils do not kill germ tubes, but reduce their …